Not A Feminist

Some random ass 25 y/o poly genderqueer kinkster:

Studies of men who buy sex (punters) show that they are significantly more likely than other men to rape and engage in all forms of violence against women. A US study found that punters were nearly eight times more likely to rape than other men.

We should not be surprised therefore that there was a marked increase in male violence against women and children after they introduced the full decriminalisation of the sex trade in New Zealand – even though it coincided with a general decrease in crime overall.

For a discussion of the data this assertion is based on, see Meme about rape in New Zealand since the full decriminalisation of the sex trade.

Sometimes it feels like even leftists don't know what capitalism is. The existence of private property ≠ capitalism. Private property has existed for thousands of years as well.

Patriarchy has thrived all throughout history, in agrarian societies, during feudalism, merkantilism, capitalism, socialism etc. Just one example: In Hungary, feudalism was only abolished in the mid-19th century. Hungary only started moving away from being an agrarian society towards capitalism in the late 19th century. Then in the 20th century, we were socialist for 40 years. Hungary as a state has existed for over 1000 years, and from those 1000 years, only ~100-150 years were spent under capitalism. But Hungarian society has remained male supremacist all the same for all 1000+ years.

A new economic system won't liberate women from male supremacy. Feminism will liberate women from male supremacy.

Leftist men will say that women are not oppressed by men, we're oppressed by capitalists, therefore we should stop criticising leftist men. Leftist white people will tell people of racial minorities that they're not oppressed by white people, they're oppressed by capitalists, so they should stop criticising leftist white people.

If I get hit by a truck, and I say, "I think part of the reason my injuries are so bad is because it was an 18-wheeler," are you going to say to me, "nooooooo it has nothing to do with the size and speed of the truck!! Don't take accountability away from the driver!!!"? Of course not.

You deny evolution when you insist that male biology has NOTHING to do with their dangerousness. It's nothing but male propaganda designed to silence women into not naming the problem. There is no reason that we cannot simultaneously acknowledge the problem AND hold men accountable -- but they've made it into a false dichotomy and you're so scared of having the wrong opinion that you fell for it. It's literally #notallmen logic repackaged with some sciencey words like "bioessentialism" to shut you up and keep your eyes closed.

The same reason men are less healthy than women is also why they're more intrinsically--biologically--evil and dangerous. The lack of a second X chromosome means there is a no backstop for a wide variety of disorders and neurocognitive impairments, many of which facilitate violence. If a female has a corrupted gene on one X chromosome, she's got a whole other one, doubling her chances of having healthy gene expression. XYs do not have this and that's why there are so many more males with profound intellectual disabilities, they die earlier and have more cancer and disease, etc. There's no backup, they're stuck with one X.

Do you know why humans have sexual dimorphism? Because your forefathers were violent rapists. If men had not been oppressing women for millennia, they would not be so much larger than us on average. We would be the same size, like many species where males and females show similar levels of aggression.

Date rape describes a forcible, coercive, or drug-assisted rape that occurs in the context of dating or partying. Marital and intimate partner rape describe forcible or coercive rape that occurs in an intimate partnership. Child sexual abuse when the victim is a child. Rape by deception is when consent was given based on intentionally false information.

"Self-rape" is not a good term and has very bad implications about the responsibility of the victim and "maintenance sex" doesn't convey how awful it is.

like if men cared about how sex is for women in these relationships or cared at all they would make sure she wanted it every time. they would be horrified at the possibility that she didn't actually want that sex act. they would stop the moment she tensed, got stiff. they wouldn't badger her. wouldn't act annoyed or indicate in any way that they expect sex and if they don't get it something bad is gonna happen. why are we pretending men are idiots who don't know when their wives/partners aren't in the mood? they know. they don't care.

men expect women to provide sex because its not a mutual act to them. ultimately it's an issue of men's entitlement to rape women they are in a relationship with. whether he scares her into saying yes out of fear of a beating or fear of him leaving, or fear that others will judge her or fear that he will think less of her. she is consenting under duress. and consent under duress is not genuine consent.

we need to teach and remind women that sex is meant to be a mutually pleasurable activity, that its supposed to be enjoyable in and of itself. it requires genuine desire for the sex act on its own. not the consequences of the sex act. women doing this don't want sex, they want him to be pleased. they want him in a better mood, they want him to stop asking. they want him not to cheat. they are exchanging sex for better behaviour. it's another transaction. the sacrifice needed to secure a good husband/boyfriend/partner and whatever additional security that provides. remember that a lot of these women cannot afford to leave, meaning they are relying on the man's income/home for themselves and their children. add in the stigma and difficulty of leaving him, the pressure to "please your man" and you can clearly see all the weight's pushing women to "consent" to sex they don't actually want.

now there's an additional pressure for women to perform their enjoyment of sex. but instead of the responsibility being on men to please her, the responsibility is still on her to pretend to be pleased, because now that's what will satisfy him. if she's stiff he'll complain of a dead bedroom and leave/cheat. so she initiates, she fakes orgasms, etc. its a performance for his ego, his emotional satisfaction and subsequent better behaviour is the exchange she gets.

^Women post things like this, propositioning childbearing as an inherent flaw and challenge to women's liberation. They claim that it's either women OR children who are deserving of respect, recognition, and protection. They never stop to ask why it is never both who are worthy of it.

The result of such a limited and bigoted mindset is overwhelmingly deplorable. The child, especially before birth, is a hinderance to tolerate at best, even if the child is wanted and chosen. The woman puts up with their existence for the sake of practicality or sentimentality. At worst, children are the problem that leads to more overt forms of patriarchal oppression.

In their eyes, either the woman is destined for great things, or she must give it up entirely to give the great things to the child. Abortion then is used as an equalizer to "protect" women from the evils that would befall them as caused by children.

But what if abortion was never about liberation from oppression, but acceptance of it?

Have you forgotten that proto-feminism and first wave feminism were created and sustained by mothers? Why is it that since the second wave of modern feminism, mothers have been left behind? Abortion was legalized well before maternity leave was enforced. Birth control with horrendous side effects were forced as the only option for avoiding pregnancy, before marital rape was outlawed. Obstetric violence was accepted as the norm by childfree women who fearmonger about birth, at the expense of normalizing midwifery led care and birth justice. Why do "progressive" feminists and red pillers both see women as inferior for their childbearing potential and status? They say the same thing, just with different words and different arguments. Either a woman is liberated/too loose, or she is shackled/dutifully impregnated.

The Madonna/Whore complex is gone, and now replaced by something far more sinister: The Slave Mother/Free Woman complex. It relies on the killing of innocent children for an evil world that they had no part in creating, and on the enslavement of childbearing women who are seen as inferior for daring to utilize their non-male biology.

You cannot expect liberation for women OR children so long as you allow oppressors to pit them against each other. I was not my mother's enemy before, during, and after birth. Neither are you. If you want to know your enemy, look at the ones who immediately open handcuffs for the woman who becomes pregnant, and mocks anyone who says they shouldn't.

So long as children have to die through abortion, infant exposure, child abuse, and trafficking as linked to women's oppression, then we will continue to see women continually shackled by their own biology, by THEIR own offspring. We will continue to see the wage gap for mothers, continue to see birthing women die from medical negligence, continue to watch women be coerced into abortions they didn't want by their community, we will continue to watch as women and children are isolated from society, culture, and political influence.

Only one group benefits from this unnatural system of subjugation, and the children never had a say in that system's formation. YOU know EXACTLY who benefits from this oppression.

My "reply" to the art above is this: Both mother and child were always destined for greatness. The people who deny this equity are the ones who want you browbeaten into subjugation.

And I know damn well feminists can do better than that.

It makes women seem pathetic. Our enemy. Our cause for oppression. Are babies. 🫩 oppressed by a baby. And if it’s not the baby (it’s a twofold issue) it’s our own biology. Well if our own biology oppresses us, why do we care about the patriarchy and misogynistic men?

–*EXTREMELY LONG INTERSEX MASTERPOST AHEAD*–

PART 1

Okay, so I’ll start this off by explaining what intersex organizations have meant when they say “spectrum of sex,” “diversity of biological sex,” or “binary ideas of sex.” These terms have been muddied and coopted to fit other groups’ campaigning without care for what they originally meant.

When we say sex is diverse and does not fit into a binary, it means that there is more than one phenotype for the male sex and more than one phenotype for the female sex. Both a male who is not intersex and someone with AIS, an XY condition that makes the body unable to use androgens and therefore no male development occurs giving the person a female phenotype, are still male. Male encompasses the spectrum of XY sexual development between non-intersex male and completely androgen-free males with female phenotypes like AIS and XY lipoid CAH people. Male also includes Swyer Syndrome, where the coding for the hormone that switches uterus development to testes development is mutated and causes uterine development instead.

As for females, you have non-intersex females and you can have females with phenotypes like mine with CAH or a male phenotype like De La Chapelle Syndrome (XX condition where during spermatogenesis a portion of a Y chromosome is accidentally fused onto the arm of the gamete’s intended X). That gives us a spectrum of the way females can be.

There is no “binary” way of being male because males can have penises and functioning testes (non-intersex), clitoromegaly and internal testes (PAIS), or a female-appearing vulva and regular clitoris size with internal testes (AIS). There are even males with uteri in the case of Swyer Syndrome. There is no “binary” way of being female because of the same things.

The “spectrum of sex” is where we overlap phenotypes between two sexes. A non-intersex female will look phenotypically the same as someone with AIS. A non-intersex male will look phenotypically the same as someone with De La Chapelle Syndrome. And I, an intersex female with androgen exposure, will look like an intersex male with PAIS who has had only partial of general male androgen exposure. A side note on that last part, for several years I thought Pidgeon Pagonis had CAH too based on the genitalia descriptions they gave because it was all the same with me. I didn’t know any better until I joined the intersex Discord and the topic came up.

However, while there are not dichotomous ways of being male or female, we know that everyone is either male or female for an important reason: the only reason ours or any species has biological sex is to add different angles and gametes to sexual reproduction. In humans, there are only two options: to make and develop a system to use ova for pregnancy, or to make and develop a system to use spermatozoa for impregnation. No intersex person produces the opposite sex’s gamete or has the full and functioning system of the opposite sex to use that gamete. Most of us are infertile.

If intersex people represented a biological sex other than male or female, we would expect most people to be intersex and few to be male or female. This is like any other thing in statistics: opposite-end extremes are the rarest, while roughly 95% falls in the middle-range, just like a bell curve.

Or, in a less numbery graphic:

We also know that truly being intersex (having a major difference of congenital reproductive development, not PCOS, hormonal differences, or general NCAH) is not 1.7% as many “activists” love to boast about, comparing to red hair and green eyes. The true statistic is about .0667% of the population having an intersex condition.

I’m going to break things down very clearly under the cut for everyone, looking at each intersex variant, how it arises, what sex, why it belongs solely to that sex, the frequency of occurrence etc. Things will be linked to sources. Some sources may overlap because I’m trying to keep each entry consistent but the information is on the same page as another part’s source, like finding the occurrence frequency. Some links will be from MedScape, which has a free registration to view more than a few links but the sources there are good–I will remain skimpy with using these links because of the needing to register. It’ll be very long so I’m giving you guys the benefit of the cut so you don’t have to scroll through miles on your dash to get to the next post. Like, I am not shitting you. Painfully, severely, agonizingly long and detailed. It has taken me more than 14 hours to compile and source this post.

If you click a link to one of the diseases associated with varying gene’s mutations for different intersex conditions and cannot find how it is tied to that mutation of the gene, you can open that gene’s protein or enzyme UniProt page as linked in its description in this post and go to the section for Pathology & Biotech. All diseases associated with intersex gene mutations that I have sourced can be found there.

Content warning for some linked (not shown outright) images of nude people, genitalia, and internal organs.

TURNER SYNDROME (45, X or 45, X0)

(Alternate general information link)

Rate of Occurrence: 1 in 2,500 girls

“This condition occurs in about 1 in 2,500 newborn girls worldwide, but it is much more common among pregnancies that do not survive to term (miscarriages and stillbirths).”

Genes Involved: Missing or incomplete second X chromosome sometimes both X chromosomes but one is rendered completely dysfunctional by mutations, karyotype written as “45, X” or “45, X0.”

Health Concerns Associated with Turner Syndrome (Note: not all patients, but many will have these, some will not have any, and many will have some but not others)

Source 1:

“About 30 percent of females with Turner syndrome have extra folds of skin on the neck (webbed neck), a low hairline at the back of the neck, puffiness or swelling (lymphedema) of the hands and feet, skeletal abnormalities, or kidney problems. One third to one half of individuals with Turner syndrome are born with a heart defect, such as a narrowing of the large artery leaving the heart (coarctation of the aorta) or abnormalities of the valve that connects the aorta with the heart (the aortic valve). Complications associated with these heart defects can be life-threatening.”

Source 2:

Before birth

“Turner syndrome may be suspected prenatally based on prenatal cell-free DNA screening ― a method to screen for certain chromosomal abnormalities in a developing baby using a blood sample from the mother ― or prenatal ultrasound. Prenatal ultrasound of a baby with Turner syndrome may show:

Large fluid collection on the back of the neck or other abnormal fluid collections (edema)

Heart abnormalities

Abnormal kidneys

                               At birth or during infancy

Signs of Turner syndrome at birth or during infancy may include:

Wide or weblike neck

Low-set ears

Broad chest with widely spaced nipples

High, narrow roof of the mouth (palate)

Arms that turn outward at the elbows

Fingernails and toenails that are narrow and turned upward

Swelling of the hands and feet, especially at birth

Slightly smaller than average height at birth

Slowed growth

Cardiac defects

Low hairline at the back of the head

Receding or small lower jaw

Short fingers and toes

                              In childhood, teens and adulthood

The most common signs in almost all girls, teenagers and young women with Turner syndrome are short stature and ovarian insufficiency due to ovarian failure that may have occurred by birth or gradually during childhood, the teen years or young adulthood. Signs and symptoms of these include:

    Slowed growth

    No growth spurts at expected times in childhood

    Adult height significantly less than might be expected for a female member of the family

    Failure to begin sexual changes expected during puberty

    Sexual development that “stalls” during teenage years

    Early end to menstrual cycles not due to pregnancy

    For most women with Turner syndrome, inability to conceive a child without fertility treatment”

OMIM Entry

Clinical reports on patients: (X) (X) (X)

Endocrine Profile: abnormally high FSH, LH, and gonadotropin

Intersex: Only occurs in females. No male forms without the presence of Wolffian-determinant coding factors exclusive to the Y chromosome. There is no Y equivalent to Turner Sydrome; humans require at least one X to survive so all fetuses conceived with a singular Y karyotype do not live to be birthed. Turner patients commonly do not form ovaries and instead the tissue degenerates into streak gonads, but in some cases where partial functioning of a second X remains, ovaries may form. Almost all Turner Syndrome patients are infertile.

Images of deformity associated with chromosomal aneuploidy of Turner Syndrome:

More severe, from complete destruction of one X or only inherited one X:

Less severe, partial inheritence of a second X or second X without complete destruction due to mutations:

Some Turner patients may have almost complete functioning of their second X and look and have the same health level as anyone else.

KLINFELTER SYNDROME (XXY)

Rate of Occurence: 1 in 500 to 1 in 1,000 boys

Genes Involved: An extra X chromosome has been placed into either the sperm or ova before conception, so the baby boy has an XXY karyotype.

Health Concerns Associated With Klinefelter Syndrome (again, not all, but a good number)

“Other physical changes associated with Klinefelter Syndrome are usually subtle. Older children and adults with the condition tend to be somewhat taller than their peers. Other differences can include abnormal fusion of certain bones in the forearm (radioulnar synostosis), curved pinky fingers (fifth finger clinodactyly), and flat feet (pes planus).

Children with Klinefelter syndrome may have weak muscle tone (hypotonia) and problems with coordination that delay the development of motor skills, such as sitting, standing, and walking. Affected boys often have learning disabilities, problems with reading, and mild delays in speech and language development. Boys and men with Klinefelter syndrome tend to have better receptive language skills (the ability to understand speech) than expressive language skills (vocabulary and the production of speech) and may have difficulty communicating and expressing themselves. They tend to have anxiety, impaired social skills, a short attention span, and limited problem-solving skills (executive functioning)

Compared with unaffected men, adults with Klinefelter syndrome have an increased risk of developing type 2 diabetes, blood clots, involuntary trembling (tremors), breast cancer (if gynecomastia develops), thinning and weakening of the bones (osteoporosis), and autoimmune disorders such as systemic lupus erythematosus and rheumatoid arthritis. (Autoimmune disorders are a large group of conditions that occur when the immune system attacks the body’s own tissues and organs.)”

OMIM Entry (Couldn’t find one for just the syndrome itself, this one is the closest and focuses on its relation to testicular tumors. Information on this is primarly found in the “Molecular Genetics” section.)

Clinical reports on patients: (X) (X) (X) (X)

Intersex: Klinefelter Syndrome only occurs in males. The intersex symptom is inevitable onset of gynecomastia, lower sperm count, smaller penis and testes size, and low facial and body hair development. As they have proper-functioning Y chromosomes and anti-Mullerian activity, they produce all Wolffian structures, no Mullerian structures, and produce spermatozoa and not ova.

Some Klinefelter guys (literally just normal guys, if you don’t believe me these all came from Google image results for ‘Klinefelter Syndrome’ and I checked the pages they were tied to to make sure they were in fact Klinefelter guys):

XXXY SYNDROME (48, XXXY)

Rate of Occurence: between 1 in 17,000 and 1 in 50,000 boys

Genes Involved: Unintended inheritance of two extra X chromosomes from a dysfunctionally created gamete.

Health Concerns Associated With XXXY Syndrome:

“48,XXXY syndrome is a chromosomal condition in boys and men that causes intellectual disability, developmental delays, physical differences, and an inability to father biological children (infertility). Its signs and symptoms vary among affected individuals.

Most boys and men with 48,XXXY syndrome have mild intellectual disability with learning difficulties. Speech and language development is particularly affected. Most affected boys and men can understand what other people say more easily than they themselves can speak. The problems with speech and communication can contribute to behavioral issues, including irritability and outbursts or temper tantrums. Boys and men with 48,XXXY syndrome tend to have anxiety, a short attention span, and impaired social skills.

48,XXXY syndrome is also associated with weak muscle tone (hypotonia) and problems with coordination that delay the development of motor skills, such as sitting, standing, and walking. Affected boys and men tend to be taller than their peers, with an average adult height of over 6 feet.

Other physical differences associated with 48,XXXY syndrome include abnormal fusion of certain bones in the forearm (radioulnar synostosis), an unusually large range of joint movement (hyperextensibility), elbow abnormalities, curved pinky fingers (fifth finger clinodactyly), and flat feet (pes planus). Affected individuals may have distinctive facial features, including widely spaced eyes (ocular hypertelorism), outside corners of the eyes that point upward (upslanting palpebral fissures), and skin folds covering the inner corner of the eyes (epicanthal folds). However, some boys and men with 48,XXXY syndrome do not have these differences in their facial features.

48,XXXY syndrome disrupts male sexual development. The penis is shorter than usual, and the testes may be undescended, which means they are abnormally located inside the pelvis or abdomen. The testes are small and do not produce enough testosterone, which is the hormone that directs male sexual development. The shortage of testosterone often leads to incomplete puberty. Starting in adolescence, affected boys and men may have sparse body hair, and some experience breast enlargement (gynecomastia). Their testes typically do not produce sperm, so most men with this condition are infertile.”

(No OMIM Entry found)

Clinical reports on patients: (X) (X) (X) <- Last link includes info about other intersex aneuplodies like XXYY and XXXYY, etc.

Intersex: Occurs in males only. The Y chromosome is there and functions, producing anti-Mullerian ability. Regardless of the number of X chromosomes, one “no” in the way of anti-Mullerian development from even a single Y causes male development. They still have Wolffian structures, may be able to perform spermatogenesis, and a male phenotype, though they are infertile. The penis may be smaller and the testes may be undescended. They never have Mullerian structures, menstruate, or produce ova.

Guys with XXXY Syndrome:

XXYY SYNDROME (48, XXYY)

Rate of Occurrence: between 1 in 18,000 and 1 in 40,000 boys

Genes Involved: Due to defects in the parents’ gamete production, inheritance of an extra X and an extra Y from a defective gamete.

Health Concerns Associated With XXYY Syndrome

“48,XXYY syndrome is a chromosomal condition that causes infertility, developmental and behavioral disorders, and other health problems in males.

48,XXYY disrupts male sexual development. Adolescent and adult males with this condition typically have small testes that do not produce enough testosterone, which is the hormone that directs male sexual development. A shortage of testosterone during puberty can lead to reduced facial and body hair, poor muscle development, low energy levels, and an increased risk for breast enlargement (gynecomastia). Because their testes do not function normally, males with 48, XXYY syndrome have an inability to father children (infertility).

48,XXYY syndrome can affect other parts of the body as well. Males with 48,XXYY syndrome are often taller than other males their age with an average adult height of 6 feet 4 inches (193 cm). They tend to develop a tremor that typically starts in adolescence and increases with age. Dental problems are frequently seen with this condition; they include delayed appearance of the primary (baby) or secondary (adult) teeth, thin tooth enamel, crowded and/or misaligned teeth, and multiple cavities. As affected males get older, they may develop a narrowing of the blood vessels in the legs, called peripheral vascular disease. Peripheral vascular disease can cause skin ulcers to form. Affected males are also at risk for developing a type of clot called a deep vein thrombosis (DVT) that occurs in the deep veins of the legs. Additionally, males with 48,XXYY syndrome may have flat feet (pes planus), elbow abnormalities, abnormal fusion of certain bones in the forearm (radioulnar synostosis), allergies, asthma, type 2 diabetes, seizures, and congenital heart defects.

Most males with 48,XXYY syndrome have an IQ that ranges from 70-80 with some degree of difficulty with speech and language development. Learning disabilities, especially those that are language-based, are very common in males with this disorder. Affected males seem to perform better at tasks focused on math, visual-spatial skills such as puzzles, and memorization of locations or directions. Some boys with 48,XXYY syndrome have delayed development of motor skills such as sitting, standing, and walking that can lead to poor coordination. Affected males have higher than average rates of behavioral disorders, such as attention deficit hyperactivity disorder (ADHD); mood disorders, including anxiety and bipolar disorder; and autism spectrum disorder, which affects communication and social interaction.”

(No OMIM Entry found)

Clinical reports on patients: (X) (X) (X)

Intersex: Again, because of the presence of at least one non-mutated Y chromosome, only males have XXYY. No Mullerian structures, no ova production. Wolffian structures are present.

Guys with XXYY Syndrome:

That should just about cover it for sex chromosome aneuploidies. Obviously we can guess by all this information that they aren’t causing a third sex and instead cause many health concerns no matter the amount of chromosomes or combinations.

XX / XY MOSAICISM

Rate of Occurence: extremely rare and unknown; actual human chimeras are incalculably rare

How this works: This is also called “tetragametic chimerism,” meaning that a baby has inherited genetics from four gametes instead of two. This occurs when a fraternal twin absorbs its twin zygote at some point in pregnancy, adding the twin’s DNA to different locations in its body, sometimes mixing the DNA sometimes not.

(I don’t particularly like this graphic but it gets the point across)

What sex then? If the gonadal tissue receives enough XX zygote DNA, the person will develop a female phenotype and sex development regardless of even being 96% XY DNA elsewhere. Sometimes, absorption of an opposite sex zygote’s DNA can cause development of ovotesticular disorder of sex development, which I’ll get into in its own section.

Here, the person was determined to be only one sex but then the opposite sex fraternal twin was absorbed. The person still only has the ability to produce and use one type of gamete, not both. It was not biology’s intention for the person to have both XX and XY cells and it does not represent nature’s desire to create a third sex. People are still phenotypically one sex or the other. You will not see a patient with a fully developed penis making ova. Most commonly the patient has mixed gonadal dysgenesis (described above in the Swyer Syndrome section of Part 1).

Health Concerns Associated With XX / XY Mosaicism

Congenital Pulmonary Lymphangiectasis with Chylothorax (behind a paywall but the title alone tells you what’s going on)

Perinatal Fatality (again, paywall, only abstract is shown)

Clinical reports on patients: (X) (X) (X) (X)

OVOTESTICULAR DISORDER OF SEX DEVELOPMENT

(60% of all patients with ovotestes are 46, XX. The remainder are XX / XY mosaic chimeras, for which refer to the above passage on XX / XY Mosaicism.)

Rate of Occurence: 1 in 83,000

Genes Involved: Most often in patients who are not mosaics, ovotesticular disorder is caused by faulty sperm genesis in which the father’s sperm production accidentally places one of his Y’s SRY regions onto the arm of another sperm’s X.

What sex? Patients with 46, XX ovotesticular disorder are all females who happened to receive male coding regions by accident and it is a “nonclassical” form of De La Chapelle Syndrome, which is described below. Most often the coding region is SRY, but it may also be caused by unnatural duplication or triplication of SOX9 or duplications or deletions in SOX3. (See sections ‘TEXT’ and ‘Genetic Heterogeneity of 46,XX Sex Reversal’)

As for mosaics with ovotesticular disorder, see their info above.

There is not a condition of ovotesticular disorder where the patient has both a fully functioning penis and vagina, and so cannot make use of both gametes. The person still only has one reproductive capability (if not rendered infertile or physically unable), either pregnancy or impregnation. Persons with ovotesticular disorder do not add a new angle to human sexual reproduction and that is most important in realizing that they are not a third biological sex.

Health Concerns Associated With Ovotesticular Disorder

Cancer of the Testicular Tissue

It is hard to find other information regarding health concerns as they would be specific to which genes are mutated, what hormone levels have been caused, and what quantity of each gonadal tissue has developed. Doctors also care less about these patients’ health and more about photographic their “freaky” patients for fame and fortune, so almost all case reports on ovotesticular disorder disregard the patient’s health in favor of talking about sexual development.

Clinical reports on patients: (X) (X) (X)

DE LA CHAPELLE SYNDROME

Rate of Occurence: unknown

Genes Involved: In 80 - 80% of cases, unnatural translocation of SRY onto an arm of an X during father’s spermatogenesis. The other cases include mutations of SOX9, SOX3, RSPO1 and WNT4, all important genes in sex development.

OMIM Entry (disregard ovotestis-related info here, as that only applies to “nonclassical” De La Chapelle Syndrome that manifests as ovotesticular disorder)

Clinical reports on patients: (X) (X)

Intersex: De La Chapelle Syndrome only occurs in females. It is intersex only because the patient is not actually male. While testes are present, they are very small and unable to produce sperm. The person does not produce ova either. Because this is a female disorder, some breast development may take place and there is lower-than-average testosterone production. The patient may be given testosterone HRT to help with prevention of gynecomastia and influence secondary sex development, if wished.

That’d cover just about all of them, I’d think. Onto our conclusion.

Do intersex people categorize a third sex or disprove that human sex is only male or female? No.

Nothing listed above shows any option toward reproduction other than pregnancy or impregnation, sperm or ova, Mullerian or Wolffian. There is no intersex variant that can make and successfully use both ova and sperm. There is no intersex variant with strict Mullerian development that makes sperm, and no intersex variant with strict Wolffian development that makes ova.

Also, we can tell that different types of being intersex are exclusively tied to mutations of certain genes, which are mutations leading to health concerns or sometimes death. If intersex were a third sex, different disorders wouldn’t be tied to desctruction of function in very specified genes and would instead be healthfully inherited development through a third sex chromosome.

Intersex disorders also tend to be specifically common in some ethnicities and not others, such as virilizing CAH in Ashkenazi Jews, lipoid CAH in Koreans, and 5ARD in the Dominican Republic, some highland tribes in New Guinea, Lebanon and Turkey. All around the world, no matter where you go, almost 50% of people are male and 50% of people are female (save for countries with high rates of femicide). If intersex people were a third sex, we would have each condition show equal rate of occurrence in all ethnicities across the board. But we do not.

We also would not be able to mix “sexes” like having both Klinefelter Syndrome (XXY) and CAH.

Biological sex in humans is dichotomous. We can only be male or female, but there is a spectrum of how males present and a spectrum of how females present. Intersex people do not disprove the existence of biological sex or prove the existence of new sexes; we merely have mutations of genes that altered our course of development, though none that truly reverse our sex and allow us to fully perform the reproductive ability of the opposite sex or both sexes at the same time. Many of us are sterile and unhealthy, and many of us have to live on pharmaceuticals to care for the poor bone and heart health that is so frequent among intersex disorders.

White men say it's brown men

Left wing men say it's right wing men

Right wing men say it's left wing men

Muslim men say it's Christian men

Christian men say it's muslim men

Poor men say it's billionare men

Billionaire men say it's poor men

Gay men say it's straight men

Straight men say it's gay men