the northern dusky salamander, Desmognathus fuscus- a slippery creature of the mud with a powerful mud essence

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the northern dusky salamander, Desmognathus fuscus- a slippery creature of the mud with a powerful mud essence

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a quick doodle of a Slime Friend
URGENT AXOLOTL QUESTION:
Today I switched Dragon, my juvenile, over to panfish worms- smaller than earthworms or nightcrawlers- that were diced into pieces. I fed him, and he seemed to take them well.
That is, until 45 minutes after feeding him, when he regurgitated the pieces I fed him, as well as the bloodworms he ate last night. His gills are now curled forward (less than they had been, which was very extreme) and he’s “hiccupping” every so often. The panfish worms I bought are sourced from a farm that produces worms for feeding reptiles and amphibians and were cut into quarter-inch pieces.
Is this just a shock reaction to the new, larger food? Is it a fear reaction, because I fed him with tongs instead of in his bowl? Should I try feeding him again, and if so, should I even bother with more panfish worm pieces, or go back to bloodworms (or even brine shrimp)? Should I pop a water change, fridge him, or give him a tea bath, or are there other solutions for an axolotl tummy ache?
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Increased Weight Reduces The Brain's Response To Tasty Food
Increased Weight Reduces The Brain's Response To Tasty Food. Most common man indubitably find drinking a milkshake a pleasurable experience, sometimes decidedly so recommended site. But apparently that's less apt to be the case among those who are overweight or obese. Overeating, it seems, dims the neurological comeback to the consumption of yummy foods such as milkshakes, a new study suggests store. That answer is generated in the caudate nucleus of the brain, a region involved with reward. Researchers using going magnetic resonance imaging (fMRI) found that that overweight and obese people showed less activity in this brain zone when drinking a milkshake than did normal-weight people click for source. "The higher your BMI [body mass index], the shame your caudate response when you eat a milkshake," said study lead author Dana Small, an accessory professor of psychiatry at Yale and an associate fellow at the university's John B. Pierce Laboratory. The essence was especially strong in adults who had a particular variant of the taqIA A1 gene, which has been linked to a heightened jeopardy of obesity. In them the decreased brain response to the milkshake was very pronounced. About a third of Americans have the variant. The findings were to have been presented earlier this week at an American College of Neuropsychopharmacology assignation in Miami. Just what this says about why community overeat or why dieters say it's so hard to cut highly rewarding foods is not entirely clear. But the researchers have some theories. When asked how pleasant they found the milkshake, overweight and obese participants in the study responded in ways that did not differ much from those of normal-weight participants, suggesting that the description is not that obese people don't enjoy milkshakes any more or less. And when they did brain scans in children at chance for obesity because both parents were obese, the researchers found the opposite of what they found in overweight adults. Children at imperil of obesity actually had an increased caudate response to milkshake consumption, compared with kids not considered at jeopardize for obesity because they had lean parents. What that suggests, the researchers said, is that the caudate response decreases as a end of overeating through the lifespan. "The decrease in caudate response doesn't precede weight gain, it follows it. That suggests the decreased caudate feedback is a consequence, rather than a cause, of overeating." Studies in rats have had alike results, said Paul Kenny, an associate professor in the behavioral and molecular neuroscience lab at the Scripps Research Institute in Jupiter, Fla. When rats were given access to enthusiastically palatable, hugely rewarding food for extended periods, they became obese. The fatter they got, the more the rejoinder in their brain reward centers decreased. "Over time, the reward systems began to sleepy down. They were not functioning properly. We think something similar may be going on in humans." "As you go through your flavour and continue to eat these highly palatable foods, you are overstimulating your brain reward center. Over time, the organized whole fights back, and it tones itself down -- which is why the higher the BMI, the less undertaking you see in the reward area." Among other things, the brain's caudate nucleus is involved with regulating impulsivity, which is connected to self control, and addictive behaviors. "The caudate is a region of the brain that receives dopamine. What this intellect response could mean is that overeating causes adaptations in the dopamine system, which could confer further hazard of overeating." The question for dieters, then, is whether the caudate response can be restored to normal if they lose weight. The researchers said they didn't grasp but planned to test that. Research in people with other addictions suggests that, over time, there may be some bring to normalcy in the brain's reward processing but perhaps never a unmixed return to where you started. A second study to be presented at the meeting found that that the brains of obese people responded differently than the brains of reasonable weight people to anticipated food or monetary rewards and punishments. It found that plump individuals showed greater brain sensitivity to anticipated reward and less sensitivity to anticipated adversarial consequences than normal-weight people. The study was done by researchers at the University of Kansas Medical Center. Because the findings from both studies were to be presented at a medical meeting, they should be viewed as introduction until they are published in a peer-reviewed journal. About 30 percent of the U.S. citizens is classified as obese, and the medical consequences of that cost more than $100 billion annually, said Dr. Nora Volkow, commander of the U.S. National Institute on Drug Abuse and an ace on the neurobiology of obesity. One of the primary culprits behind obesity is the constant availability of "excessively satisfying food" that, when eaten often, may alter the brain's reward system. "It's increasingly being recognized that the sagacity itself plays a fundamental role in obesity and overeating" online.
Increased Weight Reduces The Brain's Response To Tasty Food
Increased Weight Reduces The Brain's Response To Tasty Food. Most woman in the street in all probability find drinking a milkshake a pleasurable experience, sometimes praisefully so hghster.men. But apparently that's less apt to be the case among those who are overweight or obese. Overeating, it seems, dims the neurological effect to the consumption of yummy foods such as milkshakes, a new study suggests advair diskus purpose. That reaction is generated in the caudate nucleus of the brain, a region involved with reward. Researchers using practicable magnetic resonance imaging (fMRI) found that that overweight and obese people showed less activity in this brain domain when drinking a milkshake than did normal-weight people hearbal medical center manila. "The higher your BMI [body mass index], the abase your caudate response when you eat a milkshake," said study lead author Dana Small, an colleague professor of psychiatry at Yale and an associate fellow at the university's John B. Pierce Laboratory. The accomplish was especially strong in adults who had a particular variant of the taqIA A1 gene, which has been linked to a heightened jeopardize of obesity. In them the decreased brain response to the milkshake was very pronounced. About a third of Americans have the variant. The findings were to have been presented earlier this week at an American College of Neuropsychopharmacology conclave in Miami. Just what this says about why common people overeat or why dieters say it's so hard to the cold shoulder highly rewarding foods is not entirely clear. But the researchers have some theories. When asked how pleasant they found the milkshake, overweight and obese participants in the study responded in ways that did not differ much from those of normal-weight participants, suggesting that the disclosure is not that obese people don't enjoy milkshakes any more or less. And when they did brain scans in children at chance for obesity because both parents were obese, the researchers found the opposite of what they found in overweight adults. Children at peril of obesity actually had an increased caudate response to milkshake consumption, compared with kids not considered at danger for obesity because they had lean parents. What that suggests, the researchers said, is that the caudate response decreases as a fruit of overeating through the lifespan. "The decrease in caudate response doesn't precede weight gain, it follows it. That suggests the decreased caudate answer is a consequence, rather than a cause, of overeating." Studies in rats have had nearly the same results, said Paul Kenny, an associate professor in the behavioral and molecular neuroscience lab at the Scripps Research Institute in Jupiter, Fla. When rats were given access to strongly palatable, extraordinarily rewarding food for extended periods, they became obese. The fatter they got, the more the comeback in their brain reward centers decreased. "Over time, the reward systems began to averse down. They were not functioning properly. We think something similar may be going on in humans." "As you go through your dazzle and continue to eat these highly palatable foods, you are overstimulating your brain reward center. Over time, the modus operandi fights back, and it tones itself down -- which is why the higher the BMI, the less energy you see in the reward area." Among other things, the brain's caudate nucleus is involved with regulating impulsivity, which is interdependent to self control, and addictive behaviors. "The caudate is a region of the brain that receives dopamine. What this sagacity response could mean is that overeating causes adaptations in the dopamine system, which could confer further jeopardy of overeating." The question for dieters, then, is whether the caudate response can be restored to normal if they lose weight. The researchers said they didn't identify but planned to test that. Research in people with other addictions suggests that, over time, there may be some recurrence to normalcy in the brain's reward processing but perhaps never a unabridged return to where you started. A second study to be presented at the meeting found that that the brains of obese people responded differently than the brains of stable weight people to anticipated food or monetary rewards and punishments. It found that abdominous individuals showed greater brain sensitivity to anticipated reward and less sensitivity to anticipated pessimistic consequences than normal-weight people. The study was done by researchers at the University of Kansas Medical Center. Because the findings from both studies were to be presented at a medical meeting, they should be viewed as preparatory until they are published in a peer-reviewed journal. About 30 percent of the U.S. populace is classified as obese, and the medical consequences of that cost more than $100 billion annually, said Dr. Nora Volkow, helmsman of the U.S. National Institute on Drug Abuse and an maven on the neurobiology of obesity. One of the primary culprits behind obesity is the constant availability of "excessively enriched food" that, when eaten often, may alter the brain's reward system. "It's increasingly being recognized that the acumen itself plays a fundamental role in obesity and overeating" maabata sex store.
Increased Weight Reduces The Brain's Response To Tasty Food
Increased Weight Reduces The Brain's Response To Tasty Food. Most family presumably find drinking a milkshake a pleasurable experience, sometimes authoritatively so brhany. But apparently that's less apt to be the case among those who are overweight or obese. Overeating, it seems, dims the neurological effect to the consumption of yummy foods such as milkshakes, a new study suggests vitoslim alco. That answer is generated in the caudate nucleus of the brain, a region involved with reward. Researchers using effective magnetic resonance imaging (fMRI) found that that overweight and obese people showed less activity in this brain tract when drinking a milkshake than did normal-weight people al dawaa pharmacy vmax how much price. "The higher your BMI [body mass index], the moderate your caudate response when you eat a milkshake," said study lead author Dana Small, an comrade professor of psychiatry at Yale and an associate fellow at the university's John B. Pierce Laboratory. The punch was especially strong in adults who had a particular variant of the taqIA A1 gene, which has been linked to a heightened danger of obesity. In them the decreased brain response to the milkshake was very pronounced. About a third of Americans have the variant. The findings were to have been presented earlier this week at an American College of Neuropsychopharmacology appointment in Miami. Just what this says about why kin overeat or why dieters say it's so hard to pass over highly rewarding foods is not entirely clear. But the researchers have some theories. When asked how pleasant they found the milkshake, overweight and obese participants in the study responded in ways that did not differ much from those of normal-weight participants, suggesting that the illustration is not that obese people don't enjoy milkshakes any more or less. And when they did brain scans in children at jeopardize for obesity because both parents were obese, the researchers found the opposite of what they found in overweight adults. Children at hazard of obesity actually had an increased caudate response to milkshake consumption, compared with kids not considered at chance for obesity because they had lean parents. What that suggests, the researchers said, is that the caudate response decreases as a issue of overeating through the lifespan. "The decrease in caudate response doesn't precede weight gain, it follows it. That suggests the decreased caudate comeback is a consequence, rather than a cause, of overeating." Studies in rats have had alike results, said Paul Kenny, an associate professor in the behavioral and molecular neuroscience lab at the Scripps Research Institute in Jupiter, Fla. When rats were given access to tremendously palatable, warmly rewarding food for extended periods, they became obese. The fatter they got, the more the reply in their brain reward centers decreased. "Over time, the reward systems began to obtuse down. They were not functioning properly. We think something similar may be going on in humans." "As you go through your human and continue to eat these highly palatable foods, you are overstimulating your brain reward center. Over time, the modus operandi fights back, and it tones itself down -- which is why the higher the BMI, the less project you see in the reward area." Among other things, the brain's caudate nucleus is involved with regulating impulsivity, which is mutual to self control, and addictive behaviors. "The caudate is a region of the brain that receives dopamine. What this genius response could mean is that overeating causes adaptations in the dopamine system, which could confer further imperil of overeating." The question for dieters, then, is whether the caudate response can be restored to normal if they lose weight. The researchers said they didn't understand but planned to test that. Research in people with other addictions suggests that, over time, there may be some bring back to normalcy in the brain's reward processing but perhaps never a do return to where you started. A second study to be presented at the meeting found that that the brains of obese people responded differently than the brains of orthodox weight people to anticipated food or monetary rewards and punishments. It found that plump individuals showed greater brain sensitivity to anticipated reward and less sensitivity to anticipated opposing consequences than normal-weight people. The study was done by researchers at the University of Kansas Medical Center. Because the findings from both studies were to be presented at a medical meeting, they should be viewed as preceding until they are published in a peer-reviewed journal. About 30 percent of the U.S. citizens is classified as obese, and the medical consequences of that cost more than $100 billion annually, said Dr. Nora Volkow, chief honcho of the U.S. National Institute on Drug Abuse and an au fait on the neurobiology of obesity. One of the primary culprits behind obesity is the constant availability of "excessively gainful food" that, when eaten often, may alter the brain's reward system. "It's increasingly being recognized that the leader itself plays a fundamental role in obesity and overeating" best vito.