#scientificresearch

https://www.regulations.gov/document/OMB-2026-0034-0001

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last semester i was taking a lab focusing on tire-wear particles and found this graphical abstract

NO WAY

IT WAS HIM!!!

i just remembered these though i’m so sad i cannot find the link anywhere and i didn’t use that paper as a citation

。・゜・(ノД`)・゜・。

BONUS

here’s some non scientific memes i made while taking the lab

It will now get a bit technical, but I swear, the text gets more readable after 3 paragraphs!

ASPD is a continuation of Conduct Disorder. Clinically, Conduct Disorder is divided into Childhood Onset and Adolescence Onset. Both have distinct aetiological factors, the former closely associated with biological factors of affective cognitive functioning and physiological responses, possibly inhibiting the development of remorse, fear of punishment, and empathy, while the latter has its origin predominantly, but not exclusively, in social factors, (Daphne V Keen 2007, Silberg J, Moore AA, Rutter M 2014).

Yet, they show a very similar neurological profile and similarly impaired executive disfunctions and impulsivity as well as anti-social behavior and those with displaying anti-social behavior due a strong genetic disposition or neurological misdevelopment are always linked with abuse or neglect (Johnson VA, Kemp AH, Heard R, Lennings CJ, Hickie IB, 2015).

Furthermore, Conduct Disorder among children often leads to parental dismissal. (Paul J. Frick, Ph.D. 2024) Conduct Disorder of those with a childhood onset, often display the so-called Callous Unemotional traits (Carolina Facci 2023). This is what many lay-people mean when hey speak about "Psychopathy", the "affective" component within ASPD and Conduct Disorder (Farrah N. Golmaryami, 2021). Against some common misconceptions, these people are no less impulsive than those with adolescence conduct disorder. When we speak about the two factor model, the emotional component (factor 1) leads to the behavioral (factor 2). Factor 2 is the result of factor 1. If factor 1 does not lead to factor 2 , it is not factor 1 , but something else. In this regard, both childhood and adolescence results in the same behavior.

It is quite possible though, that the personal experience with childhood onset differs from those with adolescence onset. And maybe this also explains the accusations of misportrayal of ASPD. Someone with a less genetic early onset may have a different experience from those with an adolescence onset?

I am lucky to have met mutuals on Tumblr, two of them I really hold dear, those experience is very similar to mine. Stories we share about our childhood bear resemblences, even though the details vary. Emotional dysregulations are the same, and the advises we exchance to navigate our lives are also mutually helpful. We have similar worries and similar aspirations. You can clearly see, it is the same disorder we share.

However, all of them have a childhood onset of theri dissocial behavior. I cannot say how it is for those who aquired their anti-social traits later in life, but I can confirm that there is a shared cluster of experience between those with a childhood onset and hence, if we trust the studies, suffer from a deficit in neurological development - combined with emotional neglect and abuse. It makes me wonder, how we would have turned out f we grew up in a perfect envrionment?

Back to the question: When I speak about ASPD childhood/adolescence onset, I mean Conduct Disorder childhood/adolescence onset. Since ASPD is a continuiation of Conduct Disorder, the onset for ASPD has started either in teenagehood or childhood, depending on the factors I described in detail above.

I also read that something like an adult-onset ASPD is in discussion for those who developed anti-social traits in adulthood, but it is unclear how promising this idea is, given that a personality disorder refers to a personality presumed to persist through life. Such a drastic chance during adulthood may be something else than a personality then. But lets see how this turns out!

And if you are intersted in details about what I wrote, you can make your self a picture about that matter here. Rejoice in the sources:

Johnson VA, Kemp AH, Heard R, Lennings CJ, Hickie IB. Childhood- versus adolescent-onset antisocial youth with conduct disorder: psychiatric illness, neuropsychological and psychosocial function. PLoS One. 2015 

Silberg J, Moore AA, Rutter M. Age of onset and the subclassification of conduct/dissocial disorder. J Child Psychol Psychiatry. 2015

Farrah N. Golmaryami, Erin P. Vaughan, Paul J. Frick, Callous-unemotional traits and romantic relationships, Personality and Individual Differences, Volume 168, 2021

Fawcett C, Wesevich V, Truedsson E, Wåhlstedt C, Gredebäck G. Callous-unemotional traits affect adolescents' perception of collaboration. J Child Psychol Psychiatry. 2016

Carolina Facci, Enrico Imbimbo, Federica Stefanelli, Enrica Ciucci, Andrea Guazzini, Andrea Baroncelli, Paul J. Frick, The Social Correlates to Callous-Unemotional Traits in a Sample of High School Students, Behavior Therapy, Volume 54, Issue 3, 2023

Paul J. Frick, Ph.D, The Influence of Parenting on Callous-Unemotional Traits and the Implications for the Causes and Treatment of Conduct Disorder, American Journal of Psychiatry Volume 181, Number 4

HELLO TUMBLR !!! THIS IS MY NEUROANATOMY SITE IVE BEEN DEVELOPING FOR MY COLLEGE!!! TO OFFER FREE EDUCATION TO ALL !!!!

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Like, I know. I know being disabled makes the research all the more meaningful and it shows the world how capable we are. Not only that but it's the representation we need in the field, being disabled is a BIG part of someone too so it should make sense but.

We aren't JUST our disability. At least I don't think not to that extent. We all have wishes, dreams and motivations like other people despite our limitations. And it becomes infuriating after some time when I want to hear and study a research made by a disabled person, but all that articles are saying is less about the research and all about the researcher.

I don't think this is fair to the researcher either. I personally believe most scientists want to have their research known before people know their names. Of course being credited and recognized is important, but when that recognition goes beyond your own research, it becomes a problem.

Dark matter is one of the most fascinating mysteries of modern physics 🌌. It is an invisible form of matter that cannot be directly observed because it does not emit, absorb, or reflect light 🔭. Although we cannot detect it with ordinary telescopes, scientists are certain that it exists due to its strong gravitational influence on galaxies and the universe.

In space, stars in galaxies move in ways that cannot be explained by visible matter alone 🌠. They move much faster than expected, which suggests that an invisible "mass" is holding them all together. This invisible mass is what we call dark matter.

Dark matter does not interact with light, but it does interact with it through gravity 🪐. This makes it extremely difficult to study. Scientists believe that it makes up about 27% of the universe, while ordinary matter (such as stars, planets, and humans) only makes up about 5% 🌍.

Researchers use powerful tools like space telescopes, particle detectors, and large experiments at CERN 🔬 to search for dark matter particles. They also study galaxy clusters and cosmic radiation to find clues.

Although we still don't know what dark matter is made of, several theories suggest it could be made of unknown particles like WIMPs or axions 🧪.

Dark matter is important because it helps explain how galaxies formed and how the universe evolved over billions of years 🚀. Without it, the universe as we know it wouldn't exist.